effects of chronic exposure to hypoxia and physical training on ventilatory responsiveness..
Read Online

effects of chronic exposure to hypoxia and physical training on ventilatory responsiveness..

  • 130 Want to read
  • ·
  • 36 Currently reading

Published in [Madison] .
Written in English


  • Respiration

Book details:

Edition Notes

Series[Oregon. University. School of Health, Physical Education, and Recreation. Microform publications]
The Physical Object
Paginationv, [4], 321 l.
Number of Pages321
ID Numbers
Open LibraryOL14626164M

Download effects of chronic exposure to hypoxia and physical training on ventilatory responsiveness..


Effects of chronic hypoxia from birth on the ventilatory response to acute hypoxia in the newborn rat. Changes in carotid body and nTS neuronal excitability following neonatal sustained and chronic intermittent hypoxia exposure, Respiratory Physiology R. Harding, Development of ventilatory responsiveness to progressive hypoxia and Cited by: Exposure to chronic or intermittent hypoxia produces alterations in the ventilatory response to hypoxia. These adaptations can differ depending on the severity of the hypoxic stimulus, its duration, its pattern, and the presence or absence of other chemical by:   First, hyperoxia had profound effects on dyspnea and exercise endurance in this group of patients with chronic ventilatory insufficiency. Second, improved exercise performance was primarily related to reduced ventilatory demand, which, in turn, led to improved operational lung volumes and a delay in the attainment of limiting ventilatory Cited by: Long‐term exposure to hypoxia generates changes in respiratory control known as ventilatory acclimatization to hypoxia. The object of this study was to investigate the degree to which the hyperventilation and hypocapnia can induce the changes known as ventilatory acclimatization to hypoxia, in the absence of the primary hypoxic stimulus itself.

  This study was completed in conjunction with a study designed to examine the effects of endurance training on V ̇ O 2 peak and skeletal muscle composition in high altitude natives. Data for oxygen uptake, power output and minute volume come from that study (Favier et al., a, Favier et al., b).A group of 18 young men, residents of La Paz, Bolivia ( m) were assigned to either an.   The purpose of these studies are to elucidate the effect of training and provide the proper types of altitude/hypoxic training through the frequency of exposure, training altitude, characteristics of subjects, and types of training in the systematic review and meta-analysis 3, 4, 17, 30, However, it is still controversial whether these.   Unwanted side effects of hypobaric hypoxia include headache, chest pain associated with insufficient blood flow to the heart, palpitations, and dizziness. Mechanisms differentiating normobaric vs. hypobaric hypoxia may include differences in ventilatory patterns, alveolar gas disequilibrium, and acute hypoxic ventilatory responses. Although IH experienced by humans with OSA often occurs over years, most animal models involve exposure to chronic intermittent hypoxia (CIH) for more limited durations (4 days to 4 wk; Refs. 33, 41). For technical reasons relating to the speed at which oxygen can be exchanged in a chamber large enough to hold rodents, the frequency of hypoxic.

The present study was performed to investigate the effects of a combination of intermittent exposure to hypoxia during exercise training for short periods on ventilatory responses to hypoxia and hypercapnia (HVR and HCVR respectively) in humans. In a hypobaric chamber at a simulated altitude of 4, m (barometric pressure mmHg), seven subjects (training group) performed exercise training. Intermittent hypoxia (IH), characterized by repeated episodes of hypoxia/reoxygenation, has profound and contrasting effects on the susceptibility of the myocardium to ischemia-reperfusion injury.   Intermittent chronic hypoxia and metabolic consequences. Chronic exposure to IH as present in OSA may contribute to metabolic consequences including type 2 diabetes, insulin resistance, dyslipidemia, and obesity. The most evident factor linking metabolic consequences and IH is sympathetic overactivity 38 that increases catecholamine levels. Angiogenesis and Muscle Capillarization. In Table 2, some examples of the effects of intermittent hypoxia exposure on angiogenesis, vascular remodeling, muscle capillarization and hypertension are addition to its erythropoietic role, HIF-1 is the main mediator of angiogenesis in response to hypoxic conditions (Rey and Semenza, ) and has been considered a potential therapeutic.